Chronic inflammation comprises a vast array of diseases that affect millions of people worldwide. Chronic inflammatory diseases include asthma, arthritis, atherosclerosis, Alzheimer’s disease, autoimmune diseases and allergies, to name but a few.
A common theme to all of the above diseases is that the inflammatory response, originally aimed at recognizing and eliminating foreign “pathogens” or damaging agents, overreacts to them and causes tissue damage that can be detrimental to the patient.
How to limit such overreaction without compromising the efficiency of the inflammatory response is a phenomenal challenge faced by scientists and clinicians working in this field. Our current therapeutic tools, in particular anti-inflammatory drugs and steroids, fall short of accomplishing such a task as they can decrease the patient’s natural defence mechanisms and produce a host of undesirable side effects.
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