The growing proportion of ageing individuals in our society is affected by late onset diseases including neurodegenerative disorders.
The reasons why age is the main risk factor for neurodegenerative diseases such as Alzheimer‘s or Parkinson‘s disease are still unknown.
Are there common mechanisms in the cellular dysfunction that is associated with normal ageing and neurodegenerative disease? Are there common mechanisms between early and late onset forms of dementia and motor dysfunction and how do they relate to the processes that underlie ageing?
A better understanding of the molecular basis of both ageing and neurodegenerative diseases will likely lead us to new therapeutic and preventive strategies.
One highlight of the conference is the awarding of the 1st Hartwig Piepenbrock-DZNE prize. The prize, endowed with 60,000 Euros, is donated by the Piepenbrock Group and awarded jointly with the DZNE. It recognizes outstanding scientific contributions towards the understanding, prevention and cure of neurodegenerative disorders such as Alzheimer's and Parkinson's diseases.
Participation at the conference is free of charge for journalists. An accreditation is requested: http://www.conference-ageing-neurodegeneration.de/registration/registration-for-journalists.htm
Katrin Weigmann | idw
LaserForum 2018 deals with 3D production of components
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Proteins must be folded correctly to fulfill their molecular functions in cells. Molecular assistants called chaperones help proteins exploit their inbuilt folding potential and reach the correct three-dimensional structure. Researchers at the Max Planck Institute of Biochemistry (MPIB) have demonstrated that actin, the most abundant protein in higher developed cells, does not have the inbuilt potential to fold and instead requires special assistance to fold into its active state. The chaperone TRiC uses a previously undescribed mechanism to perform actin folding. The study was recently published in the journal Cell.
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