Three new studies by researchers at UC Davis Cancer Center provide new pieces to the puzzle of why some prostate cancers become resistant to androgen suppression therapy. The studies were presented Sunday afternoon at the 2003 annual meeting of the American Urological Association.
Of the nearly 190,000 men in the United States who develop prostate cancer every year, a substantial proportion will require androgen suppression therapy to reduce levels of male hormones -- a treatment that can shrink prostate cancers or slow their growth. Hormone suppression therapy eventually fails, however, as prostate cancer cells adapt to an androgen-depleted environment, a state known as androgen independence. When this happens, few treatment options remain.
Determining how androgen independence develops, and how the process can be derailed, is a chief focus of prostate cancer research at UC Davis. "If we could prevent androgen independence from happening, it would have a dramatic impact on treatment and outcomes for prostate cancer," says Ralph deVere White, chair of urology at UC Davis School of Medicine and Medical Center and director of the UC Davis Cancer Center.
Claudia Morain | EurekAlert!
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