A scientist at the Marine Biological Laboratory (MBL) has published the results of an EPA-funded clam embryo study that supports her hypothesis that, when combined, the pollutants bromoform, chloroform, and tetrachloroethylene—a chemical cocktail known as BCE—can act synergistically to alter a key regulator in nerve cell development. While scientists have previously studied the effects of these pollutants individually, this is the first time anyone has demonstrated that BCE’s components can work together to adversely affect neuron growth in a model organism.
The study, which is reported in the January 2005 issue of Environmental Toxicology and Pharmacology, is the first step toward understanding how exposure to BCE might affect human nerve cell development—knowledge that may one day provide clues about such neurological mysteries as autism spectrum disorder or attention deficit hyperactivity disorder.
To test her hypothesis, MBL scientist Carol Reinisch and her colleagues treated developing surf clam embryos (Spisula solidissima) with different combinations of BCE and studied their effects on nerve cell growth. “On a cellular level, clam neurons are extremely useful in studying basic mechanisms of cell development,” says Reinisch, an expert in PCB-induced neurotoxicity.
Pamela Clapp Hinkle | EurekAlert!
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