A cellular sensor of dietary fats slows the development of lesions that lead to heart disease, a Salk Institute study has found.
The study, which appears in the Oct. 17 edition of Science and is posted on the journals web site, uncovers a unique pathway that significantly curbs the development of atherosclerosis - the accumulation of fatty deposits on arterial walls. The pathway could be used to develop drugs to treat heart disease, currently the number one killer of Americans.
Ronald Evans, the March of Dimes Chair in Molecular and Developmental Biology at the Salk Institute, research fellow Chih-Hao Lee and colleagues found that the regulatory molecule, PPAR-delta (short for peroxisome proliferator-activated receptor), plays a powerful part in the bodys inflammatory response to the beginning phases of atherosclerosis.
Andrew Porterfield | EurekAlert!
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